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In vitro studies using human somatic cells suggest that Epithalon may activate telomerase , an enzyme involved in maintaining telomeres
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Moreover, ERS activates NF-κB via the IRE1-TRAF2 pathway, upregulating pro-IL-1β to prime pyroptosis, while pyroptosis-derived IL-1β feedback amplifies neuronal ERS, forming a self-sustaining death loop [221]
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