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Is oxidative stress an epiphenomenon of dysfunctional and dying neurons, or does oxidative stress itself cause the dysfunctionality/death of neurons

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The physiological level of hydrogen peroxide) (H 2 O 2 ) and nitric oxide ( ∙ NO) can selectively react with the thiol function of the cysteinyl residues at the active site of the proteins (receptors, enzymes, transporters, etc.)