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With the beneficial effect of BPC 157 therapy, this can occur along with blocking VEGF signaling during the “angiogenic switch” and initial tumor growth [95] and the counteraction of severe adverse effects occurring with cytostatic drug applications [180,181,182], thereby improving their anti-tumor effectiveness

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The logic is straightforward: cagrilintide activates amylin receptors while tirzepatide activates GLP-1 and GIP receptors