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to identify candidate genes, which when inhibited would preferentially reduce the viability of missense mut-p53 cancer cells (which typically accumulate mut-p53 protein) compared with cells that are p53 wild-type or null (non-missense p53, which typically have low or absent p53 levels)
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Funding This research was funded by the Deanship of Scientific Research at King Khalid University through the Large Groups Project under grant number R.G.P2/200/43
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