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These studies found that replenishment of glutathione by administering the glutathione precursor S -adenosyl-l-methionine (SAMe) or the use of other antioxidants attenuated alcohol-induced liver damage (see Wu and Cederbaum 2003)

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In the HF group, we observed significant upregulation of mitochondrial 3-ketoacyl-CoA thiolase (2.8-fold increase) and delta(3,5)-Delta(2,4)-dienoyl-CoA isomerase (2.3-fold increase), representing adaptive responses to increased dietary fat load

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