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TLRs bind to microbial ligands, as well as endogenous molecules (heat shock proteins [Hsps], ECM components) that signal injury and mediate the action of SCs during the inflammatory response.[20,21] Second, dedifferentiated SCs multiply and organise within the basal laminar scaffold (that spared by WD in the endoneurial tubes), to form the bands of Büngner, guiding the axonal sprouts and ensuring the neurotrophic and neurotropic support.[22] The growth cone In the proximal stump, the retrograde morphological changes that reflect nerve regeneration consist of the transformation of the stable axonal segment into a highly active growth cone
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