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Whereas the apoptosis pathway of regulated cell death is often genetically or epigenetically impaired in primary high-risk and relapsed neuroblastomas 14 , there is only limited knowledge of how metabolic rewiring as a consequence of aberrant MYCN controls the liabilities of fast-proliferating malignant cells that are confronted with accumulating reactive oxygen species (ROS)
glucoamylase
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FSH is the main factor that prevents follicles from inevitable apoptotic demise [19]
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