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The process is facilitated by the presence of Polyethylene Glycol, a molecular crowding agent which brings α-synuclein molecules in close proximity by displacing water from the immediate vicinity for enhanced hydrogen bonding, non-covalent interactions and hydrophobic interactions (De Luca et al., 2025)

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How to efficiently deliver SP to inflammatory lesions in RA in vivo is another problem that urgently needs to be solved