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BPC 157 counteracted various venous occlusion-induced syndromes[9-11], inferior caval vein syndrome[9], ischemia-reperfusion injury following Pringle maneuver[10], and Budd-Chiari syndrome[11] in rats

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Given that loss of function of PINK1 or Parkin results in symptoms of PD in humans but not mice (162), this suggests that it is a combination of the genetic mutation together with an inflammatory insult that leads to PD in individuals with mutations in PINK1/PARK2