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In RBCs, the auto-oxidation of oxyHb can lead to the formation of both O 2 •− and metHb: The production of O 2 •− could be elicited by the oxidative-dependent activation of NADPH oxidase [17], according to the reaction: The aberrant activation of NADPH oxidase may result in impaired cell deformability, metHb formation, and decreased cell survival
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