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members of the NADPH oxidase (NOX) family, the mitochondrial electron transport chain, or extracellular lysyl oxidase activity in collagen crosslinking 19,20,21,22
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Consequently, mice lacking both copies of the cytoplasmic superoxide dismutase 1 (SOD1) gene or having one copy deleted for the mitochondrial superoxide dismutase 2 (SOD2) gene exhibit noticeable oxidative harm and experience spontaneous cancer development (Gill et al., 2016)
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