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Animal data suggest improved tendon‑to‑bone healing, protection against muscle damage, and accelerated wound closure, potentially via nitric oxide pathways and pro‑angiogenic effects

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The damage appears to be upstream, at the level of the hypothalamic signal that starts the pulse (Priem, Shanley and Malan, Metabolism 1976, showing that prior consumption of ethanol attenuates the normal growth hormone response and proposing depletion of hypothalamic catecholamine stores as the mechanism)